Allergy - download pdf or read online

By S. Holgate, et. al.

ISBN-10: 0723436584

ISBN-13: 9780723436584

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Genetics of allergic disease. J Allergy Clin Immunol 2010; 125(2 suppl 2):S81–94. A positive association between the phenotype and the allele will occur if the allele is the cause of, or contributes to, the phenotype. This association would be expected to be replicated in other populations with the same phenotype, unless there are several different alleles at the same locus contributing to the same phenotype, in which case association would be difficult to detect, or if the trait was predominantly the result of different genes in the other population (genetic heterogeneity), or depended on interaction with an environmental exposure not present in the replication population.

CCR chemokine receptor family The CCR chemokine receptors are expressed on cells important to allergic inflammation including eosinophils, basophils, lymphocytes, macrophages, and dendritic cells, whereas the CXCR are expressed mainly on neutrophils and lymphocytes. Activation of chemokine cell surface receptors by specific chemokines results in activation of a cascade of intracellular signalling pathways, including guanosine triphosphate-binding proteins of the Ras and Rho families, leading ultimately to the formation of cell surface protrusions termed uropods and lamellipods, which are required for cellular locomotion.

E. vascular cell adhesion molecule-1 (VCAM-1)], as well as to receptors in the extracellular matrix (the CS-1 region of fibronectin). The α4 integrins support firm adhesion of leukocytes to VCAM-1, and can also support leukocyte rolling on endothelium in vivo. 23 1 Introduction to mechanisms of allergic disease β Ligand binding site The immunoglobulin superfamily of endothelial cell expressed adhesion molecules α Divalent cation binding sites Divalent cation binding sites NH2 Cysteine-rich repeats NH2 S S Membrane COOH Transmembrane domain COOH Cytoplasmic domains Fig.

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Allergy by S. Holgate, et. al.


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