By Elaine A. Moore
In 2001 Graves' affliction: a realistic consultant defined the explanations, prognosis, therapy and sickness process Graves' illness and different hyperthyroid problems, resembling poisonous multinodular goiter, thyroiditis, resistance to thyroid hormone, and hyperthyroidism brought on by drugs and genetic mutations. the current paintings maintains the above yet makes a speciality of next advances in disorder pathology, together with discoveries concerning the genetic, immune approach, and environmental elements that result in hyperthyroid issues; new directions for traditional therapy; and substitute and complementary clinical cures. extra sections describe distinct situations reminiscent of hyperthyroidism in being pregnant and in teenagers and temporary hyperthyroidism within the child.
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Extra resources for Advances in Graves' Disease and Other Hyperthyroid Disorders
Thyroglobulin and thyroid peroxidase antibodies, if present, are low and there may be a family history of autoimmune thyroid disease. Considered a variant of Hashimoto’s thyroiditis, it primarily occurs in young adults and may occur 3–12 weeks after delivery as an immunologic rebound from the normal immunosuppression that occurs in pregnancy. A period of hypothyroidism may follow the hyperthyroid phase and several cycles of alternating hyperthyroidism and hypothyroidism can develop. However, only a minority of women ﬁt the classic pattern.
However, only a minority of women ﬁt the classic pattern. Most women with PPT have only a thyrotoxic or a hypothyroid phase, which may become permanent in about 25 percent of patients. In PPT, thyrotoxicosis is caused by an inﬂammation-induced release of preformed thyroid hormone. Patients with PPT may occasionally go on to develop Graves’ disease. Also see the sections headed Hyperemesis gravidarum and Familial Hypersensitivity to HCG in Pregnancy described earlier in this chapter with causes of hyperthyroidism.
Choriogonadotropin-Mediated Thyrotoxicosis As described earlier in this chapter, high levels of or sensitivity to the hormone known as beta-human chorionic gonadotropin (beta–HCG) can cause hyperthyroidism. Beta-HCG is secreted in women during pregnancy and in individuals with various gonadotropin-secreting malignancies, including hydatidiform moles, choriocarcinomas, and rarely seminomas (type of testicular germ cell tumor). Hydatidiform mole refers to a rare mass or growth that forms inside the uterus at the beginning of a pregnancy.
Advances in Graves' Disease and Other Hyperthyroid Disorders by Elaine A. Moore