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By Susanne Klaus

ISBN-10: 1587060833

ISBN-13: 9781587060830

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Through regulatory elements identified in the proximal promoter of this gene. 78 Co-expression of C/EBP! and PPAR# induces marked synergy for the maintenance of the adipocyte phenotype. * (see bottom of p. 80 Recent data have brought new insights into the molecular events which take place at growth arrest and allow permanent exit from the cell cycle at the onset of terminal differentiation. PPAR∃ appears to initiate the effects of fatty acids on post-confluent mitoses, independently of the other adipogenic factors.

63,64 Even more striking, stromal-vascular cells from human WAT can be directed to undergo either adipogenesis or osteoblastogenesis, strongly suggesting that, in contrast to previous belief, adipose precursor cells are indeed (at least) bipotential cells. 65 Collectively, these observations emphasize that cell plasticity is more important than anticipated and that unipotentiality should be better and more cautiously defined in vitro as the only pathway that a precursor cell can follow in response to a given hormonal milieu.

Greenberg AS, Egan JJ, Wek SA et al. Perilipin, a major hormonally regulated adipocyte-specific phosphoprotein associated with the periphery of lipid storage droplets. J Biol Chem 1991; 266(17):11341-11346. 22. Himms-Hagen J. Brown adipose tissue and cold-acclimation. In: Trayhurn P, Nicholls DG, eds. Brown Adipose Tissue. London: Edward Arnold 1986; 214-268. 23. Cadrin M, Tolszczuk M, Guy J et al. Immunohistochemical identification of the uncoupling protein in rat brown adipose tissue. J Histochem Cytochem 1985; 33(2):150-154.

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Adipose Tissues by Susanne Klaus


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