H. P. F. Koppeschaar, Torsten Tuvemo, Peter Trainer, Philip's 38th International Symposium on Growth Hormone and Growth PDF

By H. P. F. Koppeschaar, Torsten Tuvemo, Peter Trainer, Philip Zeitler

ISBN-10: 331801446X

ISBN-13: 9783318014464

ISBN-10: 3805582552

ISBN-13: 9783805582551

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Read Online or Download 38th International Symposium on Growth Hormone and Growth Factors in Endocrinology and Metabolism: Granada, Spain, April 7-8, 2006 (Hormone Research) PDF

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Extra info for 38th International Symposium on Growth Hormone and Growth Factors in Endocrinology and Metabolism: Granada, Spain, April 7-8, 2006 (Hormone Research)

Example text

Diabetes 2001; 50: 2301– 2308. 11 Jessen N, Djurhuus CB, Jørgensen JO, Jensen LS, Møller N, Lund S, Schmitz O: Evidence against a role for insulin-signaling proteins PI 3-kinase and Akt in insulin resistance in human skeletal muscle induced by shortterm GH infusion. Am J Physiol Endocrinol Metab 2005;288:E194–E199. 12 Jørgensen JOL: Human growth hormone replacement therapy: pharmacological and clinical aspects. Endocr Rev 1991; 12: 189– 207. 13 Gotherstrom G, Svensson J, Koranyi J, Alpsten M, Bosaeus I, Bengtsson B, Johannsson G: A prospective study of 5 years of GH replacement therapy in GH-deficient adults: sustained effects on body composition, bone mass, and metabolic indices.

These cells did not produce insulin. 5 mouse embryos, beneath the kidney capsule of severe combined immunodeficient mice for 8 weeks. Insulin+ human cells organized into islet-like 5–25 cell clusters. These insulin+ cells coexpressed proinsulin and C-peptide and the key ␤cell transcription factors FOXA2, PDX-1 and ISL1. No insulin+ cells were found if the embryonic mouse tissue was not cotransplanted, but when hESCs were transferred alone, glucagon+ and amylase+ cells of hESC origin were found localized in ducts.

The © 2007 S. com/hre metabolic effects of GH on glucose homeostasis are complex and involve increased lipolysis and insulin antagonism resulting in glucose intolerance and insulin resistance [2–4]. In children with Turner syndrome [5, 6] and those born small for gestational age (SGA) [7–9], repeated administration of pharmacologic doses of recombinant GH leads to the induction of insulin resistance; in acromegalic adults chronic exposure to high GH levels predisposes to the development of T2DM [10, 11].

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38th International Symposium on Growth Hormone and Growth Factors in Endocrinology and Metabolism: Granada, Spain, April 7-8, 2006 (Hormone Research) by H. P. F. Koppeschaar, Torsten Tuvemo, Peter Trainer, Philip Zeitler


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